Research has shown that the nicotine in cigarette smoke may advance insulin resistance and lead to a condition known as Prediabetes.
These findings could clarify why smokers are at higher risk for diabetes than non-smokers. The same team of scientists was able to partially repeal nicotine’s effect on insulin on animal test subjects, by giving mice the nicotine-blunting drug mecamylamine.
One of the researchers, whom wanted to keep their anonymity, noted that although smokers tend to lose weight which can  help prevent heart disease, but not in all cases, still face a higher risk of being affected by diabetes.

 
Prior studies have revealed that smokers have a tendency to be more insulin-resistant, which leads to elevated blood-sugar levels. Some other studies have suggested that the key factor at work was nicotine’s effect on the stress hormone cortisol, since cortisol overload is known to induce insulin resistance over time.
In their study, the team of researchers gave adult mice injections of nicotine twice daily for 14 days. The mice displayed high levels of cortisol in their blood. They also began to eat less and lost some weight compared to mice that did not receive the shots, but nonetheless developed insulin resistance and prediabetes.
Researchers noted that after treating the mice with the nicotine-agonist drug mecamylamine, it blocked this process only somewhat. The results suggested that reducing tissue levels or decreasing insulin resistance may decrease heart disease seen in smokers. The researchers anticipate the in the future there will be drugs available to specifically block the effect of nicotine on tissue levels and insulin resistance.

Staff – EverythingAntiAging.com

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